Is it time to de-bunk this claim from nutrition and diet studies?
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Ironclad wrote:For those who would miss pizza-type crap seek ye out the Cyclic Ketogenic Diet, this involves a 're-feed' every 6th day for 24hrs (only) where the ratios are swapped, insulin spikes are the liver refills with glycogen .
Read, my babies, read. muhahaha etc
amused wrote:?.. Whether insulin plays a role is something that might be a minor issue and any effect it has could get lost in the larger role that calories-in and calories-out will play.
laklak wrote:They obsessively count calories, eating less at a meal in order to have ice cream, or nibbling celery sticks in order to have a bottle of wine.
NilsGLindgren wrote:Ironclad wrote:For those who would miss pizza-type crap seek ye out the Cyclic Ketogenic Diet, this involves a 're-feed' every 6th day for 24hrs (only) where the ratios are swapped, insulin spikes are the liver refills with glycogen .
Read, my babies, read. muhahaha etc
Well, Iron clad, I did - I found that it was a reference to a dietary regime for cildren with treatment resistant epilepsy, and where the efficacy was not obvious in adults. I also found that the diet (for chilren) was not without side effects, even though they were managable - managable as in, kidney stones, osteoporosis, etc.
What I did not find was the relevance to managing weight reduction. Or am I merely dense?
Just A Theory wrote:Sorry, in the abstract you linked there is no support for your dismissal of the role of dietary sugar in insulin resistance. (And, it's a study on diabetics -not healthy people).
How so?
From the article:Emphasis mine.
Furthermore, we have now increasing evidence that the adipose tissue not only produces free fatty acids that contribute to insulin resistance, but also acts as a relevant endocrine organ producing mediators (adipokines) that can modulate insulin signalling
Most studies on insulin resistance (the mechanism referred to indirectly by Taubes) are carried out in diabetic or pre-diabetic patients. This is because insulin resistance is one of the major risk factors for that disease.
Elena wrote:..
Exactly. Which is why citing studies on sugar intake and insulin resistance in diabeticsis irrelevant. That's what I was trying to point out. Citing studies on insulin resistance in diabetics is akin to citing papers on arteriolar resistance in hypertensive patients. We know that each type of resistance will have been increased before diabetes or hypertension develop. To understand, say, the pathogenesis of arteriolar resistance (specifically its dependence on dietary salt), we need to cite studies on arteriolar resistance in non-hypertensive people. Same for insulin resistance and dietary sugar: how do these variables behave in non-diabetics? The study you bring is in diabetics.
GT2211 wrote:I read about this book on Science Based Medicine a while back while searching for something else. I really have nothing to add since I have no real knowledge on the subject...but I will post a link if anyone is interested.
Why We Get Fat
Back to popcorn for me....
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