Posted: Oct 09, 2019 7:34 pm
by ZoonoTIKK
This scientist (Reubens) said sudden death syndrome in sleep is caused by damaged inner ears. he said that the brainstem plays very little role in CO2 detection compared to the ears, and if we damage both ears, we will lose 53% of our respiratory drive, and could develop fatal breathing irregularities.

His 'proof' of this was a study on mice done by Allen et al in 2011 (on NIH) where control mice were left with healthy ears and another group were injected with gentamicin into their ears. They were put to sleep and CO2 was put into their chamber. the mice with damaged ears did not respond to the excess CO2 and died but the healthy ones did not.

This study was mentioned in a peer review and it said there was a loss of neurons within the vestibular nuclei as well as hair cell loss. However I saw a news article from 2016 where he still says he thinks the hair cells in our ears are vital at telling us to breathe, so vital that if we lose them our brainstem cannot by itself keep us breathing normally. (see section 5.2)

I looked on Wikipedia and it says nothing about the inner ear being a critical chemoreceptor, and his statements are going very much against established science and neurology that concludes the brainstem is responsible for 80% of the respiratory drive and the peripheral chemoreceptors 20%, so in established science there is no room for the inner ear to take up 53% responsibility.