Jenniferocious wrote:For anyone who knows anything about the STAI scale.
I'm reviewing an study for a uni project. The study uses STAI to assess anxiety, gives them a self-administered intervention for 30 days, gives them a STAI test again, then gives a further 30 days treatment, then test them with STAI again. The results are all over the place!
What do you mean by "all over the place"? As in, each patient seemed to get better and worse over time, or the average scores of the patients increased and decreased over time?
Jenniferocious wrote:My question is, does the STAI take into considerations determinants of anxiety?
I hadn't heard of this measure before so keep in mind that my comments here are based solely on the information I can find on it in books and articles that are available online. Specifically, I found "
State-Trait Anxiety Inventory" and "
Practitioner's guide to empirically based measures of anxiety" to be pretty useful.
From what I've read, the scale does not attempt to determine the cause of the anxiety. Instead it is simply a
measure of anxiety, in the same way that the Hamilton rating scale for depression only measures the severity of depression, not the cause. It might be able to help us figure out what the cause is, as it distinguishes between state and trait anxiety which would presumably have different causes and comorbidities, but I don't think the test itself tells us what the cause is.
Jenniferocious wrote:Does the STAI indicate whether they are anxious long term or just on the day of the test?
Looking at the links I've presented above, the test is divided generally into two types of questions: how the person feels
right now, and how they feel
in general (which is the 'state' vs 'trait' distinction). This would give the researchers a measure for both short term and long term anxiety.
Jenniferocious wrote:Basically, how do the researchers know that it is their intervention that has changed the anxiety score (without the participants keeping a diary of their determinants of anxiety)?
Well that comes down to experimental methodology. For most research, randomised controlled trials are enough to establish causation, where they use one group as a control that receives no treatment to compare to the group that receives the treatment. If they successfully control for all other possible confounds (age, gender, social economic status, etc), then the only difference between the control group and the treatment group should be the intervention you've introduced - which means that any difference must be caused by the intervention.
There are other research designs which help us establish causation as well. For example, if we're looking at individual differences then we might not want to use average scores of groups, and instead we'd use a "within-subject" design, where the subject acts as their own control. This means that we take a baseline score, introduce the treatment and measure again, then remove the treatment and measure again, and then introduce the treatment again and measure again etc. So we're looking at how the individual compares to their own previous score, rather than comparing the scores of one group of people to the scores of a completely different group. If done correctly, you can determine what variable is causing the effect.
Jenniferocious wrote:Also, is state and trait scored usually given separately?
According to the book link above, the state version is administered prior to the trait version, and the test gives us an "S-anxiety" score and a "T-anxiety" score.
Sorry I don't know enough to provide any specific details! If you want to link to the particular article you're attempting to review, I should be able to discuss the research design more thoroughly to determine whether they are measuring what they say they're measuring. From what I could find on the STAI though, it seems to have good construct validity, test-retest reliability, etc, so it appears to be a decent assessment.