Spearthrower wrote:I'll reply to you Zoon as JJ's just repeating the same errors yet again.zoon wrote:
You are saying that mutations are the only real causes in evolution, while selection pressures are so negligibly weak that they do not qualify as causes, they are mere hypothetical models to be sharply distinguished from hard facts?
How, in your view, does science explain the extreme functionality of the lens in a human eye? Since mutations, which are biologically random, are the only causes in evolution, and selection pressures are so weak as to be insignificant, how did it come about that the lens of an eye focuses light on to the retina?
So I think you need to reconsider the first paragraph as it doesn't actually render what I said accurately, and consequently your 2nd paragraph leverages an erroneous idea which is definitely not relevant to what I wrote.
So let's break it down:You are saying that mutations are the only real causes in evolution, while selection pressures are so negligibly weak that they do not qualify as causes...
No, not quite.
First I think it's important to recognize that we're doing ornithology for birds here. We're talking about philosophy and discussing evolution, but the 2 don't necessarily employ the same language, or at least not consistently - one needs to be cautious about applying the language from one to the other.
Philosophically, we're talking about causes. A cause is something which produces an effect; effects don't precede causes. From that perspective, natural selection cannot be a cause as it comes after the mutation - what is being selected naturally is, in essence, that mutation with respect to a particular fitness landscape. The cause (mutation) precedes the evolutionary effect (natural selection).
But of course this isn't a single iteration: there is a history of causes and effects - the body of the organism in question is in essence a massive collection of effects. These actually constrain the possible mutations: for example, a given gene may have a million different ways it could mutate, but those possibilities are finite and constrained by the gene preceding the mutation. Still, when stuff changes, we must needs be able to call something causal, and if the mutation didn't arise, then no change would have occurred (not exactly true as most evolution is just statistical reshuffling, but let's keep it simple).
So a gene that is responsible for a given trait may be constrained in how it can mutate based on the DNA of the gene and its position in the sequence as any mutation outside of that scope may produce something instantly deleterious which never results in any effect on the population; basically, it's self-aborting. But there are other ways that mutation can be constrained. For example, the environment in which the organism lives may produce constraints on what mutations can occur; an organism living in a typical air environment might somehow (beyond my actual knowledge) gain a mutation that lets its cellular machinery use methane instead of oxygen, but as there is no available methane to breathe, it once again results in no evolution occurring as the organism doesn't survive. There are plenty of notional ways in which mutations can be constrained by their history and their environment, but neither their history nor their environment are actually producing the change causally, just constraining the possible changes.
Natural selection (and correspondingly selection pressures) is not "weak" in terms of evolution; what I meant is that it is weak in terms of considering causes because it can only constrain not produce particular effects (for the most part). If the latter were the case, then we'd all consider it Lamarckian Evolution, not Darwinian.
Onto your 2nd paragraph then:
Science explains the functionality (not particularly extreme in that regard at all) of the lens of the human eye in terms of gradual evolution, wherein small benefits accrued iteratively.
Mutations, as I've pointed out above, aren't strictly 'random' in the most wide meaning sense of the word - they are constrained by many things, not least the nature of DNA itself being comprised of 4 - and only 4 – nucleobases.
So there may well be many paths to improving sight (read "offering preferential fitness*) - and we have quite a selection of those on the planet. The ancestors of humans went down one particular path gaining gradual improvements iteratively over time. I am sure you've seen the sequences discussed here before:
Photoreceptor cell - offers some basic sense of direction & movement
Multiple cells congregated together - better sense of movement
Small depression focusing light onto the photoreceptive cells - directionality of light and movement
Deepening 'cup' - acuity in sensing light direction
Invagination - closing back up the exterior to limit photons - increases resolution and the production of an actual image
Overgrowth of transparent cells - protect the structure
Interior filled with clear liquid acting as a lens to aid in colour filtering, refraction, and formation of a clear image.
So in terms of causality, it's all just mutations, but those mutations of course must either be neutral in terms of fitness, or be beneficial in terms of fitness else the organism possessing those mutations cannot compete statistically; ratcheted iteratively through multiple generations, we end up with the hodgepodge of biological engineering known as the human eye... and it's really not that effective or efficient at all!
Finally, I should get onto talking about the Mullerian 2-step i.e.
Add a part.
Make it necessary.
But I have daddy duties to attend to. Basically, though... a structure can evolve from and be dependent on parts that existed at the time, but those parts may eventually be coopted or even removed without consequent loss of function in the contemporary structure. Regardless though, we are wandering further and further away from my area of expertise and many other people here are at least as well versed to explain such things as I am.
As far as the evolution of, for example, the eye, is concerned, mutations are effectively random? Even after allowing for the constraints within the DNA molecule, a mutation which makes the lens of an eye slightly cloudier is no less probable than a mutation which makes the lens slightly clearer; it’s the subsequent selection which provides the directionality?
I think we are not in any disagreement about the facts of evolution, it’s the language around causality which is problematic? Would you perhaps agree that causality is a concept which our evolved minds find very easy to use, but which is somewhat woolly in science; science more strictly is about correlations rather than causes? A cause is merely something which our evolved minds latch on to as an explanation for practical purposes.
It seems to me that constraints can count as causes, for example, the causes of the shape of a river’s course include both gravity and the topology of the terrain. In constraining the direction of the water, the topology has an effect on the course of the river, it is one of the causes shaping the river’s geography. I would say by analogy that it’s not only the mutations, but also the fitness landscape (which was there before the selection process), which should be counted in among causes of the evolved eye or living part, if the causal story is not to give a one-sided picture?
I think what’s bothering me in this particular discussion is that an insistence that mutations are causes in evolution while selection pressures (or fitness landscapes) are not, seems to me to leave a wide-open goal for creationists to exploit? If an atheist says that the only causes in evolution are mutations, and that fitness landscapes do not have causal power, then it seems to me that a creationist could happily say: “Yes! You are acknowledging that the only scientifically discernable causes in evolution are mutations, mutations are effectively random for biological purposes, therefore science has no explanation for the functionality which is so evident, for example, in eyes, therefore God.” I would be worried if I could only answer that by trying to explain that fitness landscapes aren’t powerful causes even though, over evolutionary timescales, they have highly significant effects?